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0 · Preclinical Rationale for the Use of Crizanlizumab (SEG101) in
1 · Metastatic Prostate Cancer
2 · Meeting Abstract: 2023 ASCO Annual Meeting I
3 · Dr. med. Celine Wilke – Global Program Clinical Head
4 · Celine Wilke's research works
5 · Celine Wilke Profiles
6 · Author notes
7 · Abstract
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View the profiles of people named Celine Wilke. Join Facebook to connect with Celine Wilke .
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Sehen Sie sich das Profil von Dr. med. Celine Wilke auf LinkedIn, einer professionellen .Celine Wilke's 21 research works with 1,910 citations and 3,459 reads, including: Time Course .Celine Wilke's 4 research works with 254 citations and 183 reads, including: Preclinical . Abstract. Background: PIK3CA (encoding phosphatidylinositol 3-kinase [PI3K] .
Celine Wilke, Osvaldo Mirante, Michael J. Morris, Karim Fizazi, Multivariable . The bone marrow (BM) and spleen from MF patients contain great numbers of .
Celine Wilke, MD, Valerie Campello-Iddison, MD, Anna Rita Migliaccio, PhD. . This analysis of VISION data sought to build predictive models for clinical .
Celine Wilke, MD, Angela Zhang, PhD, Florian H. Heidel, MD. Blood (2020) 136 (Supplement .
View the profiles of people named Celine Wilke. Join Facebook to connect with Celine Wilke and others you may know. Facebook gives people the power to..Celine Wilke's 21 research works with 1,910 citations and 3,459 reads, including: Time Course and Management of Key Adverse Events During the Randomized Phase 3 SOLAR-1 Study of PI3K.
Celine Wilke's 4 research works with 254 citations and 183 reads, including: Preclinical Rationale for the Use of Crizanlizumab (SEG101) in Myelofibrosis. Abstract. Background: PIK3CA (encoding phosphatidylinositol 3-kinase [PI3K] p110α) is frequently altered in cancer, resulting in PI3K/mammalian target of rapamycin (mTOR) pathway activation, and contributing to targeted therapy resistance and disease progression. Celine Wilke, Osvaldo Mirante, Michael J. Morris, Karim Fizazi, Multivariable models of outcomes with [177Lu]Lu-PSMA-617: analysis of the phase 3 VISION trial, . The bone marrow (BM) and spleen from MF patients contain great numbers of impaired megakaryocytes (MK) which have been hypothesized to be responsible for their clinical manifestation by expressing on their surface high levels of the adhesion receptor P-selectin which triggers a process of pathological neutrophil emperipolesis leading to increase.
Celine Wilke, MD, Valerie Campello-Iddison, MD, Anna Rita Migliaccio, PhD. Blood (2020) 136 (Supplement 1): 26–27. https://doi.org/10.1182/blood-2020-133896. Split-Screen. Share. Tools. Myelofibrosis (MF), the end stage of Philadelphia-negative Myeloproliferative Neoplasms, is still an unmet clinical need. This analysis of VISION data sought to build predictive models for clinical outcomes after 177 Lu-PSMA-617. Methods: In VISION, adults with PSMA+ mCRPC received 177 Lu-PSMA-617 (7.4 GBq every 6 weeks, ≤6 cycles) plus protocol-permitted standard of .
Preclinical Rationale for the Use of Crizanlizumab (SEG101) in
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celine wilke|Celine Wilke's research works
